Can we cause regression of coronary atherosclerosis?

Research output: Contribution to journalReview article

1 Citation (Scopus)

Abstract

With the advent of effective antiatherosclerotic therapies, especially lipid lowering agents, cardiovascular morbidity and mortality rates associated with coronary atherosclerosis can be reduced. A growing body of evidence suggests such therapies can retard the progression of coronary atherosclerosis and with aggressive treatment regimens can cause regression. Antiatherosclerotic, and especially lipid lowering therapies, have the potential to become an alternative to invasive interventions. This report examines clinical studies that have addressed the regression of human coronary atherosclerosis by medical therapy.

Original languageEnglish (US)
Pages (from-to)368-374
Number of pages7
JournalAmerican Journal of the Medical Sciences
Volume335
Issue number5
DOIs
StatePublished - Jan 1 2008
Externally publishedYes

Fingerprint

Coronary Artery Disease
Lipids
Cardiovascular Agents
Therapeutics
Morbidity
Mortality

All Science Journal Classification (ASJC) codes

  • Medicine(all)

Cite this

Can we cause regression of coronary atherosclerosis? / Alsafwah, Shadwan; Ramanathan, K.

In: American Journal of the Medical Sciences, Vol. 335, No. 5, 01.01.2008, p. 368-374.

Research output: Contribution to journalReview article

@article{dc6e901548d348f99d0e9983b1d1f2b0,
title = "Can we cause regression of coronary atherosclerosis?",
abstract = "With the advent of effective antiatherosclerotic therapies, especially lipid lowering agents, cardiovascular morbidity and mortality rates associated with coronary atherosclerosis can be reduced. A growing body of evidence suggests such therapies can retard the progression of coronary atherosclerosis and with aggressive treatment regimens can cause regression. Antiatherosclerotic, and especially lipid lowering therapies, have the potential to become an alternative to invasive interventions. This report examines clinical studies that have addressed the regression of human coronary atherosclerosis by medical therapy.",
author = "Shadwan Alsafwah and K Ramanathan",
year = "2008",
month = "1",
day = "1",
doi = "10.1097/MAJ.0b013e31815c3b32",
language = "English (US)",
volume = "335",
pages = "368--374",
journal = "American Journal of the Medical Sciences",
issn = "0002-9629",
publisher = "Lippincott Williams and Wilkins",
number = "5",

}

TY - JOUR

T1 - Can we cause regression of coronary atherosclerosis?

AU - Alsafwah, Shadwan

AU - Ramanathan, K

PY - 2008/1/1

Y1 - 2008/1/1

N2 - With the advent of effective antiatherosclerotic therapies, especially lipid lowering agents, cardiovascular morbidity and mortality rates associated with coronary atherosclerosis can be reduced. A growing body of evidence suggests such therapies can retard the progression of coronary atherosclerosis and with aggressive treatment regimens can cause regression. Antiatherosclerotic, and especially lipid lowering therapies, have the potential to become an alternative to invasive interventions. This report examines clinical studies that have addressed the regression of human coronary atherosclerosis by medical therapy.

AB - With the advent of effective antiatherosclerotic therapies, especially lipid lowering agents, cardiovascular morbidity and mortality rates associated with coronary atherosclerosis can be reduced. A growing body of evidence suggests such therapies can retard the progression of coronary atherosclerosis and with aggressive treatment regimens can cause regression. Antiatherosclerotic, and especially lipid lowering therapies, have the potential to become an alternative to invasive interventions. This report examines clinical studies that have addressed the regression of human coronary atherosclerosis by medical therapy.

UR - http://www.scopus.com/inward/record.url?scp=45549091600&partnerID=8YFLogxK

UR - http://www.scopus.com/inward/citedby.url?scp=45549091600&partnerID=8YFLogxK

U2 - 10.1097/MAJ.0b013e31815c3b32

DO - 10.1097/MAJ.0b013e31815c3b32

M3 - Review article

VL - 335

SP - 368

EP - 374

JO - American Journal of the Medical Sciences

JF - American Journal of the Medical Sciences

SN - 0002-9629

IS - 5

ER -